Thursday, February 24, 2011

Obstructing LOXL2 enzyme cut cancer spread dramatically

LOXL2 is an amino acid creation enzyme in the body that helps create collagen and elastin. A group of researchers in the United Kingdom have found an interesting fact. Blocking LOXL2 acids blocks cancer spread. About 90 percent of cancer fatalities are caused by cancer spreading from one part of the body to another.

Keep breast cancer from spreading by obstructing enzymes

In medical journal Cancer Research, researchers at the Institute of Cancer Research in the U.K. published their recent findings. Researchers put chemicals in cancer of the breast cells. This was so that the LOXL2 enzyme would be blocked. The body did not get the cancer cells spread to it as often when the enzyme blockers were there. The cancer that spreads quickly from breast tissue to the bloodstream is linked to LOXL2 enzymes and LOXL2 gene in the bloodstream. Cancer will end up quite much anywhere in the body after it had spread from tissue to the blood which makes death more likely.

Exactly what the LOXL2 enzyme is

Copper is used by the LOXL2 gene. This gets collagen and elastin both synthesized. The gene allows the body to create. Rebuilding takes place in the process too. In order to get cancer to move from muscle and fat and to the blood, the enzyme is used. Individuals who have cancer that spread easily typically have a high present of LOXL2 enzymes. Mice have been the only animals tested on when blocking enzymes to try and stop cancer spread. Humans haven't had the LOXL2 used yet. It has not commonly been used anyway.

The correct cancer medicine creation

Researchers have been working overtime to find a cure for cancer. Mostly specific cancers have been researched in this. Getting targeted cancer medications put together can be hard to do. This is because sometimes they differ from person to person. Up to 2 years would be needed to get FDA approval on human LOXL2 enzyme medications. This would be after the three years needed to make the medications work.

Articles cited

HC2D

hc2d.co.uk/content.php?contentId=17644



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